insulin receptor in vivo Search Results


igf-1 receptor  (Valentis Inc)
90
Valentis Inc igf-1 receptor
Igf 1 Receptor, supplied by Valentis Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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autodock vina v.1.1.2  (AUTODOCK GmbH)
90
AUTODOCK GmbH autodock vina v.1.1.2
Autodock Vina V.1.1.2, supplied by AUTODOCK GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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insulin insulin like growth factor receptor daf 2  (Thermo Fisher)
98
Thermo Fisher insulin insulin like growth factor receptor daf 2
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igf-1 receptor antagonist h-1356  (Bachem)
90
Bachem igf-1 receptor antagonist h-1356
Igf 1 Receptor Antagonist H 1356, supplied by Bachem, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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package irr  (RStudio)
90
RStudio package irr
Package Irr, supplied by RStudio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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insulin receptor  (Abcam)
99
Abcam insulin receptor
Insulin Receptor, supplied by Abcam, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/insulin+receptor+in+vivo/antibody+receptor/pmc06547494-162-14-18
Average 99 stars, based on 1 article reviews
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anti–insulin receptor β-subunit (anti-irβ  (Becton Dickinson)
90
Becton Dickinson anti–insulin receptor β-subunit (anti-irβ
Effects of shRNA-expressing adenoviruses on GSIS and the proliferation of MIN6 cells. A : MIN6 cells were infected with adenoviruses integrating shVector, shPh1, shPh2, shPh3, or shIA2 for 72 h. The expression levels of endogenous phogrin and IA-2 proteins were determined by immunoblotting with specific antibodies for phogrin and IA-2. α-Tubulin is a nontargeting control. B : MIN6 cells infected with Ad-shVec, shPh1, shPh2, or shPh3 were immunostained with anti-phogrin antibody. C : MIN6 cells expressing shPh3 were fixed at time points 24, 48, and 72 h. Cells were then immunostained with anti-phogrin and <t>anti-insulin</t> antibodies. D : The infected cells were incubated for 2 h in modified KRB (2 mmol/l glucose) before high-glucose stimulation. After stimulation for 30 min with 2 or 25 mmol/l glucose, culture media ( left ; secretion) and cells ( right ; content) were collected and subjected to RIA for insulin. Data are given as means ± SE of five independent experiments. E : Time course of MIN6 cell proliferation 24–96 h after infection with Ad-shVec, shPh1, shPh2, shPh3, or shIA2. Cell number was measured as indicated in research design and methods . Phogrin-KD or IA2-KD cells grew more slowly than the shVec-infected cells (* P < 0.001, ** P < 0.005). F : Cells infected by Ad-shVec or shPh3 for 72 h were assayed for BrdU incorporation by immunofluorescence analysis. Experiments were performed four times ( n = 12). (Please see http://dx.doi.org/10.2337/db08-0970 for a high-quality digital representation of this figure.)
Anti–Insulin Receptor β Subunit (Anti Irβ, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/insulin+receptor+in+vivo/anti+insulin/pmc02646067-4-26-35
Average 90 stars, based on 1 article reviews
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pe anti human cd220 (insr  (Becton Dickinson)
90
Becton Dickinson pe anti human cd220 (insr
Effects of shRNA-expressing adenoviruses on GSIS and the proliferation of MIN6 cells. A : MIN6 cells were infected with adenoviruses integrating shVector, shPh1, shPh2, shPh3, or shIA2 for 72 h. The expression levels of endogenous phogrin and IA-2 proteins were determined by immunoblotting with specific antibodies for phogrin and IA-2. α-Tubulin is a nontargeting control. B : MIN6 cells infected with Ad-shVec, shPh1, shPh2, or shPh3 were immunostained with anti-phogrin antibody. C : MIN6 cells expressing shPh3 were fixed at time points 24, 48, and 72 h. Cells were then immunostained with anti-phogrin and <t>anti-insulin</t> antibodies. D : The infected cells were incubated for 2 h in modified KRB (2 mmol/l glucose) before high-glucose stimulation. After stimulation for 30 min with 2 or 25 mmol/l glucose, culture media ( left ; secretion) and cells ( right ; content) were collected and subjected to RIA for insulin. Data are given as means ± SE of five independent experiments. E : Time course of MIN6 cell proliferation 24–96 h after infection with Ad-shVec, shPh1, shPh2, shPh3, or shIA2. Cell number was measured as indicated in research design and methods . Phogrin-KD or IA2-KD cells grew more slowly than the shVec-infected cells (* P < 0.001, ** P < 0.005). F : Cells infected by Ad-shVec or shPh3 for 72 h were assayed for BrdU incorporation by immunofluorescence analysis. Experiments were performed four times ( n = 12). (Please see http://dx.doi.org/10.2337/db08-0970 for a high-quality digital representation of this figure.)
Pe Anti Human Cd220 (Insr, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/insulin+receptor+in+vivo/anti+cd220/pmc06280402-22-23-26
Average 90 stars, based on 1 article reviews
pe anti human cd220 (insr - by Bioz Stars, 2026-09
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anti insr antibody  (Bioss)
93
Bioss anti insr antibody
Effects of shRNA-expressing adenoviruses on GSIS and the proliferation of MIN6 cells. A : MIN6 cells were infected with adenoviruses integrating shVector, shPh1, shPh2, shPh3, or shIA2 for 72 h. The expression levels of endogenous phogrin and IA-2 proteins were determined by immunoblotting with specific antibodies for phogrin and IA-2. α-Tubulin is a nontargeting control. B : MIN6 cells infected with Ad-shVec, shPh1, shPh2, or shPh3 were immunostained with anti-phogrin antibody. C : MIN6 cells expressing shPh3 were fixed at time points 24, 48, and 72 h. Cells were then immunostained with anti-phogrin and <t>anti-insulin</t> antibodies. D : The infected cells were incubated for 2 h in modified KRB (2 mmol/l glucose) before high-glucose stimulation. After stimulation for 30 min with 2 or 25 mmol/l glucose, culture media ( left ; secretion) and cells ( right ; content) were collected and subjected to RIA for insulin. Data are given as means ± SE of five independent experiments. E : Time course of MIN6 cell proliferation 24–96 h after infection with Ad-shVec, shPh1, shPh2, shPh3, or shIA2. Cell number was measured as indicated in research design and methods . Phogrin-KD or IA2-KD cells grew more slowly than the shVec-infected cells (* P < 0.001, ** P < 0.005). F : Cells infected by Ad-shVec or shPh3 for 72 h were assayed for BrdU incorporation by immunofluorescence analysis. Experiments were performed four times ( n = 12). (Please see http://dx.doi.org/10.2337/db08-0970 for a high-quality digital representation of this figure.)
Anti Insr Antibody, supplied by Bioss, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/insulin+receptor+in+vivo/Insulin+Receptor+Polyclonal+Antibody/pm41171889-318-43-46
Average 93 stars, based on 1 article reviews
anti insr antibody - by Bioz Stars, 2026-09
93/100 stars
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phosphorylated angiotensin ii substrate  (AnaSpec)
90
AnaSpec phosphorylated angiotensin ii substrate
Effects of shRNA-expressing adenoviruses on GSIS and the proliferation of MIN6 cells. A : MIN6 cells were infected with adenoviruses integrating shVector, shPh1, shPh2, shPh3, or shIA2 for 72 h. The expression levels of endogenous phogrin and IA-2 proteins were determined by immunoblotting with specific antibodies for phogrin and IA-2. α-Tubulin is a nontargeting control. B : MIN6 cells infected with Ad-shVec, shPh1, shPh2, or shPh3 were immunostained with anti-phogrin antibody. C : MIN6 cells expressing shPh3 were fixed at time points 24, 48, and 72 h. Cells were then immunostained with anti-phogrin and <t>anti-insulin</t> antibodies. D : The infected cells were incubated for 2 h in modified KRB (2 mmol/l glucose) before high-glucose stimulation. After stimulation for 30 min with 2 or 25 mmol/l glucose, culture media ( left ; secretion) and cells ( right ; content) were collected and subjected to RIA for insulin. Data are given as means ± SE of five independent experiments. E : Time course of MIN6 cell proliferation 24–96 h after infection with Ad-shVec, shPh1, shPh2, shPh3, or shIA2. Cell number was measured as indicated in research design and methods . Phogrin-KD or IA2-KD cells grew more slowly than the shVec-infected cells (* P < 0.001, ** P < 0.005). F : Cells infected by Ad-shVec or shPh3 for 72 h were assayed for BrdU incorporation by immunofluorescence analysis. Experiments were performed four times ( n = 12). (Please see http://dx.doi.org/10.2337/db08-0970 for a high-quality digital representation of this figure.)
Phosphorylated Angiotensin Ii Substrate, supplied by AnaSpec, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/insulin+receptor+in+vivo/phosphorylated+angiotensin+ii+substrate/pm15519225-67-1-35
Average 90 stars, based on 1 article reviews
phosphorylated angiotensin ii substrate - by Bioz Stars, 2026-09
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insulin-like growth factor 1  (InsuLine Medical Ltd)
90
InsuLine Medical Ltd insulin-like growth factor 1
Effects of shRNA-expressing adenoviruses on GSIS and the proliferation of MIN6 cells. A : MIN6 cells were infected with adenoviruses integrating shVector, shPh1, shPh2, shPh3, or shIA2 for 72 h. The expression levels of endogenous phogrin and IA-2 proteins were determined by immunoblotting with specific antibodies for phogrin and IA-2. α-Tubulin is a nontargeting control. B : MIN6 cells infected with Ad-shVec, shPh1, shPh2, or shPh3 were immunostained with anti-phogrin antibody. C : MIN6 cells expressing shPh3 were fixed at time points 24, 48, and 72 h. Cells were then immunostained with anti-phogrin and <t>anti-insulin</t> antibodies. D : The infected cells were incubated for 2 h in modified KRB (2 mmol/l glucose) before high-glucose stimulation. After stimulation for 30 min with 2 or 25 mmol/l glucose, culture media ( left ; secretion) and cells ( right ; content) were collected and subjected to RIA for insulin. Data are given as means ± SE of five independent experiments. E : Time course of MIN6 cell proliferation 24–96 h after infection with Ad-shVec, shPh1, shPh2, shPh3, or shIA2. Cell number was measured as indicated in research design and methods . Phogrin-KD or IA2-KD cells grew more slowly than the shVec-infected cells (* P < 0.001, ** P < 0.005). F : Cells infected by Ad-shVec or shPh3 for 72 h were assayed for BrdU incorporation by immunofluorescence analysis. Experiments were performed four times ( n = 12). (Please see http://dx.doi.org/10.2337/db08-0970 for a high-quality digital representation of this figure.)
Insulin Like Growth Factor 1, supplied by InsuLine Medical Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/insulin+receptor+in+vivo/insuline+like+growth+factor+1/pmc04779621-63-20-30
Average 90 stars, based on 1 article reviews
insulin-like growth factor 1 - by Bioz Stars, 2026-09
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insulin sigma aldrich cat  (Alomone Labs)
93
Alomone Labs insulin sigma aldrich cat
Effects of shRNA-expressing adenoviruses on GSIS and the proliferation of MIN6 cells. A : MIN6 cells were infected with adenoviruses integrating shVector, shPh1, shPh2, shPh3, or shIA2 for 72 h. The expression levels of endogenous phogrin and IA-2 proteins were determined by immunoblotting with specific antibodies for phogrin and IA-2. α-Tubulin is a nontargeting control. B : MIN6 cells infected with Ad-shVec, shPh1, shPh2, or shPh3 were immunostained with anti-phogrin antibody. C : MIN6 cells expressing shPh3 were fixed at time points 24, 48, and 72 h. Cells were then immunostained with anti-phogrin and <t>anti-insulin</t> antibodies. D : The infected cells were incubated for 2 h in modified KRB (2 mmol/l glucose) before high-glucose stimulation. After stimulation for 30 min with 2 or 25 mmol/l glucose, culture media ( left ; secretion) and cells ( right ; content) were collected and subjected to RIA for insulin. Data are given as means ± SE of five independent experiments. E : Time course of MIN6 cell proliferation 24–96 h after infection with Ad-shVec, shPh1, shPh2, shPh3, or shIA2. Cell number was measured as indicated in research design and methods . Phogrin-KD or IA2-KD cells grew more slowly than the shVec-infected cells (* P < 0.001, ** P < 0.005). F : Cells infected by Ad-shVec or shPh3 for 72 h were assayed for BrdU incorporation by immunofluorescence analysis. Experiments were performed four times ( n = 12). (Please see http://dx.doi.org/10.2337/db08-0970 for a high-quality digital representation of this figure.)
Insulin Sigma Aldrich Cat, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/insulin+receptor+in+vivo/Native+mouse+NGF+2%2E5S+protein/pm36476861-308-230-235
Average 93 stars, based on 1 article reviews
insulin sigma aldrich cat - by Bioz Stars, 2026-09
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Image Search Results


Effects of shRNA-expressing adenoviruses on GSIS and the proliferation of MIN6 cells. A : MIN6 cells were infected with adenoviruses integrating shVector, shPh1, shPh2, shPh3, or shIA2 for 72 h. The expression levels of endogenous phogrin and IA-2 proteins were determined by immunoblotting with specific antibodies for phogrin and IA-2. α-Tubulin is a nontargeting control. B : MIN6 cells infected with Ad-shVec, shPh1, shPh2, or shPh3 were immunostained with anti-phogrin antibody. C : MIN6 cells expressing shPh3 were fixed at time points 24, 48, and 72 h. Cells were then immunostained with anti-phogrin and anti-insulin antibodies. D : The infected cells were incubated for 2 h in modified KRB (2 mmol/l glucose) before high-glucose stimulation. After stimulation for 30 min with 2 or 25 mmol/l glucose, culture media ( left ; secretion) and cells ( right ; content) were collected and subjected to RIA for insulin. Data are given as means ± SE of five independent experiments. E : Time course of MIN6 cell proliferation 24–96 h after infection with Ad-shVec, shPh1, shPh2, shPh3, or shIA2. Cell number was measured as indicated in research design and methods . Phogrin-KD or IA2-KD cells grew more slowly than the shVec-infected cells (* P < 0.001, ** P < 0.005). F : Cells infected by Ad-shVec or shPh3 for 72 h were assayed for BrdU incorporation by immunofluorescence analysis. Experiments were performed four times ( n = 12). (Please see http://dx.doi.org/10.2337/db08-0970 for a high-quality digital representation of this figure.)

Journal: Diabetes

Article Title: Gene Silencing of Phogrin Unveils Its Essential Role in Glucose-Responsive Pancreatic β-Cell Growth

doi: 10.2337/db08-0970

Figure Lengend Snippet: Effects of shRNA-expressing adenoviruses on GSIS and the proliferation of MIN6 cells. A : MIN6 cells were infected with adenoviruses integrating shVector, shPh1, shPh2, shPh3, or shIA2 for 72 h. The expression levels of endogenous phogrin and IA-2 proteins were determined by immunoblotting with specific antibodies for phogrin and IA-2. α-Tubulin is a nontargeting control. B : MIN6 cells infected with Ad-shVec, shPh1, shPh2, or shPh3 were immunostained with anti-phogrin antibody. C : MIN6 cells expressing shPh3 were fixed at time points 24, 48, and 72 h. Cells were then immunostained with anti-phogrin and anti-insulin antibodies. D : The infected cells were incubated for 2 h in modified KRB (2 mmol/l glucose) before high-glucose stimulation. After stimulation for 30 min with 2 or 25 mmol/l glucose, culture media ( left ; secretion) and cells ( right ; content) were collected and subjected to RIA for insulin. Data are given as means ± SE of five independent experiments. E : Time course of MIN6 cell proliferation 24–96 h after infection with Ad-shVec, shPh1, shPh2, shPh3, or shIA2. Cell number was measured as indicated in research design and methods . Phogrin-KD or IA2-KD cells grew more slowly than the shVec-infected cells (* P < 0.001, ** P < 0.005). F : Cells infected by Ad-shVec or shPh3 for 72 h were assayed for BrdU incorporation by immunofluorescence analysis. Experiments were performed four times ( n = 12). (Please see http://dx.doi.org/10.2337/db08-0970 for a high-quality digital representation of this figure.)

Article Snippet: Anti-adaptin, anti–carboxypeptidase E (anti-CPE), anti–calcium-dependent activator protein (anti-CAPS), anti-munc18, anti-dynamin, anti–clathrin heavy chain, anti–phosphatidylinositol 3-kinase (anti-PI3K), anti-Grb2, and anti–extracellular signal–related kinase (anti-ERK) mouse monoclonal antibodies and anti–insulin receptor β-subunit (anti-IRβ) rabbit polyclonal antibodies were from BD Biosciences.

Techniques: shRNA, Expressing, Infection, Western Blot, Incubation, Modification, BrdU Incorporation Assay, Immunofluorescence